HIV-1 Vpr Reprograms CLR4<sup>DCAF1</sup> E3 Ubiquitin Ligase to Antagonize Exonuclease 1-Mediated Restriction of HIV-1 Infection
ABSTRACT Viral accessory proteins hijack host cell E3 ubiquitin ligases to antagonize innate/intrinsic defenses and thereby provide a more permissive environment for virus replication. Human immunodeficiency virus type 1 (HIV-1) accessory protein Vpr reprograms CRL4DCAF1 E3 to antagonize select post...
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Autores principales: | , , , , , , , , , |
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Formato: | article |
Lenguaje: | EN |
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American Society for Microbiology
2018
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Acceso en línea: | https://doaj.org/article/56ce2447f8324b7eadd44ac5a495e2e4 |
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