PHF5A promotes colorectal cancerprogression by alternative splicing of TEAD2

Dysregulated alternative splicing (AS) plays critical roles in driving cancer progression, and the underlying mechanisms remain largely unknown. Here, we demonstrated that PHF5A, a component of U2 small nuclear ribonucleoproteins, was frequently upregulated in colorectal cancer (CRC) samples and ass...

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Auteurs principaux:	Yue Chang, Yulu Zhao, Liya Wang, Meijuan Wu, Chenglong He, Mengxi Huang, Zengjie Lei, Jiahe Yang, Siqi Han, Bibo Wang, Yanyan Chen, Chao Liu, Hongju Yu, Lijun Xue, Jian Geng, Yanan Chen, Tingting Dai, Lili Ren, Qian Wang, Xiaobei Liu, Xiaoyuan Chu, Cheng Chen
Format:	article
Langue:	EN
Publié:	Elsevier 2021
Sujets:	alternative splicing PHF5A colorectal cancer TEAD2 therapeutic target Therapeutics. Pharmacology RM1-950
Accès en ligne:	https://doaj.org/article/6cfb8d68c577471dba8182838bbac540
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PHF5A promotes colorectal cancerprogression by alternative splicing of TEAD2

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