IL-1β, IL-23, and TGF-β drive plasticity of human ILC2s towards IL-17-producing ILCs in nasal inflammation

IL-1β, IL-23, and TGF-β drive plasticity of human ILC2s towards IL-17-producing ILCs in nasal inflammation

Innate lymphoid cells (ILCs) play critical immunological roles including immune surveillance at mucosal sites. Here the authors show that during nasal inflammation pathogen-induced cytokine production guides the differentiation of ILCs.

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Autores principales: Korneliusz Golebski, Xavier R. Ros, Maho Nagasawa, Sophie van Tol, Balthasar A. Heesters, Hajar Aglmous, Chantal M. A. Kradolfer, Medya M. Shikhagaie, Sven Seys, P. W. Hellings, Cornelis M. van Drunen, Wytske J. Fokkens, Hergen Spits, Suzanne M. Bal
Formato: article
Lenguaje:EN
Publicado: Nature Portfolio 2019
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Science
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Acceso en línea:https://doaj.org/article/6ea86cb26d194858951641d2dc4ea3bd
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https://doaj.org/article/6ea86cb26d194858951641d2dc4ea3bd

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