GIRK2 splice variants and neuronal G protein-gated K+ channels: implications for channel function and behavior

GIRK2 splice variants and neuronal G protein-gated K+ channels: implications for channel function and behavior

Abstract Many neurotransmitters directly inhibit neurons by activating G protein-gated inwardly rectifying K+ (GIRK) channels, thereby moderating the influence of excitatory input on neuronal excitability. While most neuronal GIRK channels are formed by GIRK1 and GIRK2 subunits, distinct GIRK2 isofo...

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Autores principales: Ezequiel Marron Fernandez de Velasco, Lei Zhang, Baovi N. Vo, Megan Tipps, Shannon Farris, Zhilian Xia, Allison Anderson, Nicholas Carlblom, C. David Weaver, Serena M. Dudek, Kevin Wickman
Formato: article
Lenguaje:EN
Publicado: Nature Portfolio 2017
Materias:
Medicine
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Science
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Acceso en línea:https://doaj.org/article/71558560ec304bf5a09b60698ab9c399
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https://doaj.org/article/71558560ec304bf5a09b60698ab9c399

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