Reactive oxygen species as a trigger of the infarct-limiting effects of early hypoxic preconditioning rat myocardium
It is believed that reactive oxygen species (ROS) are involved in the performance of this adaptive hepatoprotective effect phenomenon. However, the role of ROS in the signaling mechanism of the early hypoxic preconditioning (early HP) and the contribution of individual types of oxygen radicals in th...
Saved in:
Main Authors: | A. S. Sementsov, A. V. Krylatov |
---|---|
Format: | article |
Language: | RU |
Published: |
Scientific Сentre for Family Health and Human Reproduction Problems
2016
|
Subjects: | |
Online Access: | https://doaj.org/article/8f25aeb362b14ca2a6c4f2f9f02e11e5 |
Tags: |
Add Tag
No Tags, Be the first to tag this record!
|
Similar Items
-
PRECONDITIONING AS PROTECTION AGAINST ISCHEMIC MYOCARDIAL INJURY
by: M. G. Shurygin, et al.
Published: (2013) -
Role of reactive oxygen species in bradykinin-induced proliferation of vascular smooth muscle cells
by: VELARDE,VICTORIA, et al.
Published: (2004) -
Inhibition of peroxidase activity and scavenging of reactive oxygen species by astilbin isolated from Dimorphandra mollis (Fabaceae, Caesalpinioideae)
by: PETACCI,FERNANDO, et al.
Published: (2010) -
High-performance reoxygenation from PLGA-PEG/PFOB emulsions: a feedback relationship between ROS and HIF-1α
by: Wang J, et al.
Published: (2018) -
Upregulation of liver inducible nitric oxide synthase following thyroid hormone preconditioning: suppression by N-acetylcysteine
by: FERNÁNDEZ,VIRGINIA, et al.
Published: (2009)