Trimethylamine-N-Oxide Aggravates Kidney Injury via Activation of p38/MAPK Signaling and Upregulation of HuR

Trimethylamine-N-Oxide Aggravates Kidney Injury via Activation of p38/MAPK Signaling and Upregulation of HuR

Background: Trimethylamine-N-oxide (TMAO) is an intestinal metabolic toxin, which is produced by gut flora via metabolizing high-choline foods. TMAO is known to increase the risk of atherosclerosis and cardiovascular events in chronic kidney disease (CKD) patients. Objectives: The objective of this...

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Autores principales: Yunshi Lai, Haie Tang, Xinrong Zhang, Zhanmei Zhou, Miaomiao Zhou, Zheng Hu, Fengxin Zhu, Lei Zhang, Jing Nie
Formato: article
Lenguaje:EN
Publicado: Karger Publishers 2021
Materias:
trimethylamine-n-oxide
inflammatory injury
p38/mapk pathway
human antigen r
chronic kidney disease
Dermatology
RL1-803
Diseases of the circulatory (Cardiovascular) system
RC666-701
Diseases of the genitourinary system. Urology
RC870-923
Acceso en línea:https://doaj.org/article/adaae32e43ac43308ea3ab58749d9180
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https://doaj.org/article/adaae32e43ac43308ea3ab58749d9180

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