DisA Restrains the Processing and Cleavage of Reversed Replication Forks by the RuvAB-RecU Resolvasome

DNA lesions that impede fork progression cause replisome stalling and threaten genome stability. <i>Bacillus subtilis</i> RecA, at a lesion-containing gap, interacts with and facilitates DisA pausing at these branched intermediates. Paused DisA suppresses its synthesis of the essential c...

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Autores principales: Carolina Gándara, Rubén Torres, Begoña Carrasco, Silvia Ayora, Juan C. Alonso
Formato: article
Lenguaje:EN
Publicado: MDPI AG 2021
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Acceso en línea:https://doaj.org/article/baa81b4f188b4ffa8959324883e7bcf4
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