DisA Restrains the Processing and Cleavage of Reversed Replication Forks by the RuvAB-RecU Resolvasome
DNA lesions that impede fork progression cause replisome stalling and threaten genome stability. <i>Bacillus subtilis</i> RecA, at a lesion-containing gap, interacts with and facilitates DisA pausing at these branched intermediates. Paused DisA suppresses its synthesis of the essential c...
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Formato: | article |
Lenguaje: | EN |
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MDPI AG
2021
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Acceso en línea: | https://doaj.org/article/baa81b4f188b4ffa8959324883e7bcf4 |
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