Abnormal mitochondrial L-arginine transport contributes to the pathogenesis of heart failure and rexoygenation injury.
<h4>Background</h4>Impaired mitochondrial function is fundamental feature of heart failure (HF) and myocardial ischemia. In addition to the effects of heightened oxidative stress, altered nitric oxide (NO) metabolism, generated by a mitochondrial NO synthase, has also been proposed to im...
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| Autores principales: | , , , , , , , , |
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| Formato: | article |
| Lenguaje: | EN |
| Publicado: |
Public Library of Science (PLoS)
2014
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| Materias: | |
| Acceso en línea: | https://doaj.org/article/f11909e9fbd049feb59ddde30b0a8a64 |
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