Circulating miR-103a-3p contributes to angiotensin II-induced renal inflammation and fibrosis via a SNRK/NF-κB/p65 regulatory axis

Angiotensin II is known to cause renal inflammation and fibrosis. Here Lu et al. show that levels of circulating miR-103a-3p are elevated in hypertensive nephropathy patients and in an animal model of angiotensin II-induced renal dysfunction, and that miR-103a-3p suppresses SNRK expression leading t...

Descripción completa

Guardado en:
Detalles Bibliográficos
Autores principales: Qiulun Lu, Zejun Ma, Ye Ding, Tatiana Bedarida, Liming Chen, Zhonglin Xie, Ping Song, Ming-Hui Zou
Formato: article
Lenguaje:EN
Publicado: Nature Portfolio 2019
Materias:
Q
Acceso en línea:https://doaj.org/article/fb3cafbacbca473e82a1356b6b780951
Etiquetas: Agregar Etiqueta
Sin Etiquetas, Sea el primero en etiquetar este registro!